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G Protein-Coupled Receptors in Health and Disease, Part A
By: TaoImprint: Academic Press
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G protein-coupled receptors (GPCRs) transduce signals from a diverse array of endogenous ligands, including ions, amino acids, nucleotides, lipids, peptides, and large glycoprotein hormones. They are also responsible for our sensing of exogenous stimul
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| Title of eBook: G Protein-Coupled Receptors in Health and Disease, Part A | |
| Release Date: 09-01-2009 | |
| Publisher: Academic Press |
This eBook download is available in the following formats:
| Parent title | G Protein-Coupled Receptors in... |
|---|---|
| Encrypted (DRM) | Yes |
| SKU | 9780080911953 |
| File size | 4611 |
| Security | n/a |
| Printing | Not allowed |
| Copying | Not allowed |
| Read aloud | No Sys requirements Download reader |
| Devices | Samsung Tablet, Apple Ipad & Iphone, Barnes & Noble Nook, Kobo eReader, Aluratek Libre, Iliad, Nokia, Blackberry, Hanlin |
| Note | Excellent navigation features are available via Adobe such as bookmarks and a quick access table of contents. Text search is easily accessible. An Adobe DRM-protected file is different than a pdf file in that it uses Adobe DRM (Digital Rights Management) technology, which authors and publishers use to protect their content from illegal online distribution and to set certain privileges such as restrictions on copying and printing. |
G Protein-Coupled Receptors in Health and Disease, Part A
Chapter One
RhodopsinMediated Retinitis PigmentosaKatherine M. Malanson and Janis Lem
I. Introduction 2 II. Rhodopsin Function 3 A. Retina Structure 3 B. Rod CellSpecific Expression of Rhodopsin 5 C. Classification of Rhodopsin Mutations 7 III. Mechanisms of Rod Degeneration 9 A. P23H, VPP: A Model for Misfolded and Mislocalized Rhodopsin Mutants 9 B. P347S: A Model for a Nonautonomous Pathway and Destabilized Rhodopsin Mutants 12 C. K296E: A Model for Persistent Signaling and Alternative Signaling Pathways 16 IV. Mechanisms of Cone Degeneration 18 A. Trophic Factors 18 B. Toxic Factors 20 C. Metabolic Support 21 V. Treatments 22 A. Gene Therapy 22 B. Vitamin A and Pharmacological Supplementation 25 C. Transplantation of Stem Cells or Retinal Sheets 26 VI. Conclusions 27 References
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